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This is the least settled part of the account: a broad transcriptional signature is easy to report and hard to attribute to a specific mechanism, and the causal chain from copper binding to the observed expression changes has not been closed

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Dihexa reversed learning and memory deficits at concentrations as low as 10^-13 M (picomolar range), approximately 10 million times more potent than BDNF on a molar basis for promoting synaptogenesis